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| [[File:IMG0103.jpg|left|225x225px]]Bruxism is often reduced to dental and occlusal factors, but this overlooks the crucial role of the trigeminal nervous system. This chapter emphasizes that conventional terms like bruxism may not adequately represent the complexities of the conditions they describe.
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| Bruxism is characterized as a non-functional oral activity, distinct from eating or speaking. Despite its prevalence, it often goes unnoticed until significant dental wear occurs. This raises questions about whether dental wear can occur without bruxism.
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| The concept of 'Thegosis' suggests that bruxism might serve a physiological function to increase masticatory efficiency and muscular strength, challenging the distinction between physiological and pathological states. This perspective necessitates a reassessment of how bruxism is perceived and managed in clinical settings.
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| Bruxism has diverse etiologies, including psychological stress, physiological anomalies, and especially the involvement of the trigeminal nervous system. Traditional views of bruxism as primarily a dental or occlusal issue do not account for its neurological dimensions, which are critical for effective management.
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| Recent studies on trigeminal motoneurons suggest that bruxism may be linked to decreased inhibitory control within the trigeminal network. This emerging understanding opens new avenues for comprehending and treating bruxism beyond conventional dental interventions, emphasizing the importance of integrating neurobiological research into clinical practice.
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| Bruxism may involve complex neurophysiological processes, including the activation and modulation of trigeminal motoneurons. Studies indicate that bruxism could be related to specific neuronal discharges in areas like the raphe nuclei and the locus coeruleus, which modulate sleep and arousal states that impact muscle activity.
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| These findings suggest that bruxism could be more than a response to dental misalignment or stress but also a manifestation of broader neurophysiological activities. Understanding these processes could lead to more targeted therapies addressing the neurological components of bruxism, offering relief for patients unresponsive to conventional treatments.
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| Integrating neurophysiological insights into bruxism management can significantly benefit treatment protocols. Future research should explore the interactions between neuronal circuits and bruxism behavior to develop effective interventions. Integrating neurobiological insights with clinical practice is expected to improve patient outcomes, providing a holistic treatment approach.
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| Reevaluating bruxism through neurology underscores the need for a nuanced understanding. It challenges traditional boundaries between physiological and pathological states, suggesting bruxism is a complex neurophysiological phenomenon requiring a comprehensive diagnostic and treatment approach, incorporating the latest clinical practices and ongoing research into neural mechanisms.{{ArtBy|
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| | autore = Gianni Frisardi | | | autore = Gianni Frisardi |
| | autore2 = | | | autore2 = |