(Created page with "====<translate><!--T:93--> The partition of causal relevance</translate>==== :Always be <math>n</math> the number of people we have to conduct the analyses upon, if we divide (based on certain conditions as explained below) this group into <math>k</math> subsets <math>C_i</math> with <math>i=1,2,\dots,k</math>, a cluster is created that is called a "partition set" {{:Draft:2514}} <translate><!--T:95--> where with the symbolism <math>C_i \subset n </math> it indicates...")
 
 
Line 142: Line 142:
{{:Draft:2515}}
{{:Draft:2515}}
*
*
{{q2|<translate><!--T:107--> A homogeneous partition provides what we are used to calling Differential Diagnosis</translate>.|}}
{{bib}}

Latest revision as of 09:55, 22 September 2022

The partition of causal relevance

Always be the number of people we have to conduct the analyses upon, if we divide (based on certain conditions as explained below) this group into subsets with , a cluster is created that is called a "partition set"

: [1]

[2]

where with the symbolism it indicates that the subclass is contained in .

The partition , in order for it to be defined as a partition of causal relevance, must have these properties:

  1. For each subclass the condition must apply ie the probability of finding in the subgroup a person who has the symptoms, clinical signs and elements belonging to the set . A causally relevant partition of this type is said to be homogeneous.
  2. Each subset must be 'elementary', i.e. it must not be further divided into other subsets, because if these existed they would have no causal relevance.

Now let us assume, for example, that the population sample , to which our good patient Mary Poppins belongs, is a category of subjects aged 20 to 70. We also assume that in this population we have those who present the elements belonging to the data set which correspond to the laboratory tests mentioned above and precisa in 'The logic of classical language'.

Let us suppose that in a sample of 10,000 subjects from 20 to 70 we will have an incidence of 30 subjects showing clinical signs and . We preferred to use these reports for the demonstration of the probabilistic process because in the literature the data regarding clinical signs and symptoms for Temporomandibular Disorders have too wide a variation as well as too high an incidence in our opinion.[3][4][5][6][7][8]


An example of a partition with presumed probability in which TMJ degeneration (Deg.TMJ) occurs in conjunction with Temporomandibular Disorders (TMDs) would be the following:

[9] <translate> where</translate>
<translate> where</translate>
[10] <translate> where</translate>
<translate> where</translate>
Bibliography & references
  1. Nota 1 prima sez
  2. Nota 2 prima sez
  3. Pantoja LLQ, De Toledo IP, Pupo YM, Porporatti AL, De Luca Canto G, Zwir LF, Guerra ENS, «Prevalence of degenerative joint disease of the temporomandibular joint: a systematic review», in Clin Oral Investig, 2019».
    PMID:30311063
    DOI:10.1007/s00784-018-2664-y 
  4. De Toledo IP, Stefani FM, Porporatti AL, Mezzomo LA, Peres MA, Flores-Mir C, De Luca Canto G, «Prevalence of otologic signs and symptoms in adult patients with temporomandibular disorders: a systematic review and meta-analysis», in Clin Oral Investig, 2017».
    PMID:27511214
    DOI:10.1007/s00784-016-1926-9 
  5. Bonotto D, Penteado CA, Namba EL, Cunali PA, Rached RN, Azevedo-Alanis LR, «Prevalence of temporomandibular disorders in rugby players», in Gen Dent».
    PMID:31355769 
  6. da Silva CG, Pachêco-Pereira C, Porporatti AL, Savi MG, Peres MA, Flores-Mir C, De Luca Canto G, «Prevalence of clinical signs of intra-articular temporomandibular disorders in children and adolescents: A systematic review and meta-analysis», in Am Dent Assoc, 2016». - PMCID:26552334
    DOI:10.1016/j.adaj.2015.07.017 
  7. Gauer RL, Semidey MJ, «Diagnosis and treatment of temporomandibular disorders», in Am Fam Physician, 2015».
    PMID:25822556 
  8. Kohlmann T, «Epidemiology of orofacial pain», in Schmerz, 2002».
    PMID:12235497
    DOI:10.1007/s004820200000 
  9. Nota 1 seconda sez
  10. Nota 2 seconda sez